[1] Qureshi AI, Ling GS, Khan J. Quantitative analysis of injured, necrotic, and apoptotie cells in a new experimental model of intracerebral hemorrhage [J]. Cfit Care Med 2001, 29(01). [2] Del-Bigio MR, Yan HJ, Xue M. [J]. Journal of the Neurological Sciences: Official Bulletin of the World Federation of Neurology 2001. [3] Xue M, Del Bigio MR. Intracortical hemorrhage injury in rats:relationship between blood fractions and brain cell death [J]. Stroke 2000, 31. [4] Gong C, Hoff JT, Keep RF. Acute inflammatory reaction following experimental intracerebral hemorrhage in rat. [J]. Brain research 2000, 1(1). [5] Rosenberg GA, Mun-Bryce S, Wesley M. Collagenase-induced intraeerebral hemorrhage in rats [J]. Stroke 1990, 21. [6] Xue M, Del Bigio-MR. Intracerebral injection of autologous whole blood in rats: time course of inflammation and cell death. [J]. Neuroscience Letters: An International Multidisciplinary Journal Devoted to the Rapid Publication of Basic Research in the Brain Sciences 2000, 3(3). [7] Xue M, Del Bigio MR. Acute tissue damage after injections of thrombin and plasmin into rat striatum. [J]. Stroke: A Journal of Cerebral Circulation 2001, 9(9). [8] Mayne M, Ni W, Yan HJ, Xue M, Johnston JB, Del Bigio MR, Peeling J, Power C. Antisense oligodeoxynucleotide inhibition of tumor necrosis factor-alpha expression is neuroprotective after intracerebral hemorrhage. [J]. Stroke: A Journal of Cerebral Circulation 2001, 1(1). [9] Kraus J, Oschmann P, Lei S. High concentrations of sVCAM-1 and sICAM-1 in the eerebrospinal fluid of patients with intracerebral heamorrhage are associated with poor outcome [J]. Nouril Neurosurg Psychiatry 2002, 73. |